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GLP-1 medications work through a fundamentally different mechanismone that reduces appetite at the brain level rather than adding metabolic stress

This process, which could be stimulated under conditions of stress, envisages a chloroplastic sequence of reactions involving H 2 O 2 -triggered S -glutathionylation of diverse available protein cysteine residues, followed by regeneration of the free cysteines by glutathione-dependent GRX (Zaffagnini et al., 2012)