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In light of these intracellular observations (Kokame et al, 1996), we hypothesize that changes in the intracellular redox state associated with elevated homocysteine levels can alter the expression of multiple genes involved in other specific cellular functions that could be partially responsible for vascular disorders associated with moderate hyperhomocysteinemia

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Long-term effects are still being researched, so extended use should be approached carefully

Set point resistance : Your body defends against further weight loss through hormonal shifts, including changes in leptin, ghrelin, and insulin sensitivity, that increase hunger and slow metabolism