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showed that curcumin effectively counteracts doxorubicin (DOX)-induced neurotoxicity by suppressing autophagy while reducing oxidative and endoplasmic reticulum stress, with these protective effects mediated through activation of the Keap1-Nrf2-ARE signaling pathway

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Lin and colleagues added that there is still a large addressable population for obesity treatment, with less than 6% of our study population receiving GLP-1 receptor agonists or surgery. There were some study limitations, which included the analysis cross-sectional design and possible confounding from variations in patient adherence to GLP-1 receptor agonists

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