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GSTO1 knockdown as well as the small-molecule GSTO1 inhibitor C1-27 also caused downregulation of the Wnt inhibitor Dickkopf 1 ( DKK1 ), thombospondin 1 ( THBS1 ), the cysteine-rich angiogenic inducer CYR61 and caveolae scaffolding proteins CAV1 and CAV2 , among others

By knocking out the Uox gene, these mice completely lose the ability to degrade uric acid, resulting in a significant spike in serum uric acid levels and the spontaneous formation of hyperuricemia and urate nephropathy phenotypes [6-7]

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