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Preclinical findings revealed that AMPK the brain from seizure-induced neuronal death through induction of Bcl-2modifying factor (Bmf) normally interacts with the cytoskeleton, but upon certain cellular stresses, such as loss of extracellular matrix adhesion or energy crisis, Bmf relocalizes to mitochondria, where it can promote Bax activation and mitochondrial dysfunction (Moran et al
Future research should focus on developing strategies for selective targeting to minimize effects on normal tissues, understanding mechanisms of resistance and ways to overcome them, and optimizing combination therapies with chemotherapy, radiotherapy, or immunotherapy to enhance efficacy
Frontela C, Ros G, Martnez C, Snchez?Siles LM, Canali R, et al
