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Administration of CCK, in humans and animals, inhibits food intake by reducing meal size and its duration.17,18 Recent work suggests that CCK-A receptors expressed by vagal afferent neurons are an important target for CCK in producing sensations of satiety, as well as inhibiting gastric emptying and stimulating pancreatic digestive enzyme secretion.19 In animals it is thought that the reduction in food intake may be mediated by a paracrine/neurocrine effect since high concentrations of CCK occur only locally to the site of release.20 Thus, locally released CCK may increase vagal tone, without a significant increase in plasma CCK level

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Insomnia was reported in some rare instances, and it is thought to be related indirectly to gastrointestinal discomfort or changes in the participants metabolism

A detailed history should explore the timeline of ED onset relative to semaglutide initiation, severity and progression of symptoms, presence of morning erections, relationship factors, and psychological stressors