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Jill Carnahan, MD Wow, that was an incredible whirlwind

This system entraps the active agent within a lipid-based vesicle, enhancing its stability during transit through the GI tract and improving its absorption by protecting the active agent from degradation, controlling its release, and promoting its passage across the intestinal barrier

Unlike deficiency states, which produce well-characterised neurological and haematological symptoms, there is no clearly defined clinical syndrome associated with B12 excess from supplementation alone