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It is characterized by frameshifting or nonsense mutations in the dystrophin protein gene, which is essential for maintaining the membrane integrity of muscle fibers 265
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Observed outcomes include specific cellular and molecular endpoints such as: Enhanced fibroblast migration capacity Increased collagen and elastin deposition Modulation of pro-inflammatory and antioxidant mediators Altered transcriptional profiles related to tissue homeostasis, repair signaling, and stress response All reported observations derive exclusively from preclinical laboratory and animal studies and remain model-specific
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