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In addition to blunted monoaminergic neurotransmission, it was also shown that deficiency in neurotrophic factor effects, disturbances in neurogenesis and synaptogenesis, chronic neuroinflammatory processes, and dysregulation of the hypothalamicpituitaryadrenal (HPA) axis can largely contribute to the manifestation of depressive symptoms in patients [3,4,5]

Studies indicate that MOTS-C may: Activate AMPK pathways Improve energy metabolism Reduce oxidative stress Regulate metabolic homeostasis Because mitochondrial dysfunction is frequently observed in ASD, peptides influencing mitochondrial activity are a particularly promising area of research