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High dietary fat induces NADPH oxidase-associated oxidative stress and inflammation in rat cerebral cortex
These include a relative or absolute increase in glucagon secretion from alpha cells of the pancreas, elevated serum free fatty-acid concentrations, an impaired incretin effect (e.g., decreased secretion of glucagon-like peptide-1 [GLP-1]), enhanced renal reabsorption of glucose, and dysregulation of appetite control via decreased neurotransmitter response in the brain
The intestines are the second biggest organ that uses glucose, following the brain
mechanistic and clinical work establishing dual GIP/GLP-1 receptor agonism as a distinct pharmacological class [1][9]
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