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By stimulating PDH in cancer cells to normalize glucose metabolism, it can restore their sensitivity to anoikis and weaken their metastatic potential, suggesting that PDKs are potential targets for anti-metastasis therapy (261)

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Immunoblotting also revealed residual ExtraMito particles in mEPs, corroborating the presence of mitochondria within particles resistant to MACS-mediated depletion

( f ) Quantification of H2AX foci in cells transduced with shRNA targeting IDH1 alone or combined with overexpression of wildtype IDH1 (wtIDH1 OE) (n = 3 technical replicates from 3 independent experiments in each cell line)