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2025;4(8):EVIDoa2500173

The molecular landscape of conventional melanomas in children mimics the UV-induced mutational spectrum found in melanomas in adults, including driver mutation in the RAS/RAF/mitogen-activated protein kinase pathway, alterations in the phosphatase and tensin homolog ( PTEN ) tumor suppressor gene and, notably, transcription- activating mutations of the telomerase reverse transcriptase ( TERT ) promoter ( TERT-p ) resulting in increased TERT expression [18]

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