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As noted, GliT has also been demonstrated to play a key role in self-protection from gliotoxin (Schrettl et al., 2010) and its absence in egtA 26933 might be expected to result in sensitivity to gliotoxin

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Elevated levels of GC also promote the accumulation of visceral fat, which can exacerbate the peripheral inflammatory response triggered by the lipid dysregulation (123), which suggests that there may be different pathophysiological mechanisms between the depressive subtypes and provides clinical clues to differentiate the depressive subtypes