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They found that patients with psychosis had less of the antioxidant glutathione in their brains

The lack of clear GSH differences suggests that alterations in cerebral GSH metabolism may not be a prominent factor in the pathophysiology of autism (or at least, not one that persists throughout life), even though previous research(Bjrklund, Meguid, et al., 2020

8 Differential gene expression analysis for endoneurial macrophages in DRGs of autoantibody injected mice

Melatonins ability to maintain mitochondrial function may protect against both cuproptosis and ferroptosis, which involve mitochondrial dysfunction [12, 44, 45]