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The real question for cagrilintide isnt whether it beats tirzepatide solo, but whether its mechanism earns it a complementary role: either as part of a combination like CagriSema or as a standalone option for people who need something outside the GLP-1 family

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[14] The identity of the receptor or receptors that mediate these E-peptide effects remains, as of the current literature, unresolved and, as the in-vivo work discussed below shows, the claim of full IGF-1R independence is itself contested by studies in which E-peptide-driven hypertrophy did require the receptor