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and metabolic efficiency studies

Lidocaine is extensively metabolized in the liver, so drugs that decrease hepatic blood flow (e.g., propranolol) or inhibit CYP1A2/CYP3A4 (e.g., fluvoxamine, ketoconazole, clarithromycin) may elevate systemic concentrations and increase the risk of central nervous system or cardiovascular toxicity

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They designed a microfluidic device able to support the growth of patient-derived ISEMFs and ECs, demonstrating the response of the system to different physiological parameters, such as oxygen tension, cell density, growth factors