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2024;16:e68063

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Similarly, ROS-mediated damage to ETC complexes decreases OXPHOS efficiency, forcing the heart to rely on less efficient metabolic pathways ( In myocardial samples from HF patients, glutathione peroxidase (GPx) activity is reduced by 52% compared with controls, and this reduction is inversely correlated with elevated plasma levels of 8-isoprostanes, a marker of oxidative stress ( 2+ ATPase(SERCA2a) and dysfunctional ryanodine receptors, has been documented in failing human hearts, providing a mechanistic basis for impaired excitationcontraction coupling ( 3.4 Mitochondrial membrane potential imbalance In the context of HF, the ma critical bioelectrical gradient essential for maintaining mitochondrial function and myocardial energy metabolismis significantly disrupted ( Myocardial biopsy samples from patients with dilated cardiomyopathy demonstrate reduced m together with diminished complex I and IV activities

As research continues to evolve, medical professionals must balance patient interest in cutting-edge interventions with the need for evidence-based practice