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Rhodes, D

These effects are likely tied to glucagon receptor signaling, which drives fat oxidation and reduces lipid synthesis in hepatic tissue

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Although ANGPTL4 does not directly activate TGF- receptors, it significantly amplifies cellular profibrotic signaling through mechanisms such as activating the dipeptidyl peptidase-4 (DPP-4) pathway and upregulating pro-inflammatory cytokines (Srivastava et al., 2024)