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In an initial attempt to transform the highly active AngIV analog, Nle 1 -AngIV, into an effective drug, we evaluated the procognitive activity of a series of C-terminal truncated peptides derived from Nle 1 -AngIV (Benoist et al., 2011)

The process can be slowed with the addition of glutathione, indicating that inhibition of proteins by this mechanism may rely on redox status [88]

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These two properties together are exceedingly rare in neuroprotective compounds and open the door to practical administration protocols that do not require injections or intranasal delivery