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Some research has suggested that this effect is mediated through BPC-157s interaction with growth hormone receptor pathways and its angiogenic support of the periosteum, the vascular tissue layer surrounding bone that is essential for early-stage bone repair

This receptor modulation triggers a cascade of intracellular signaling events that ultimately result in the activation of hormone-sensitive lipase, the enzyme responsible for breaking down stored triglycerides into free fatty acids and glycerol

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