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demonstrated that Tom20 or Bax knockout effectively blocks ROS-induced pyroptosis, while antioxidant treatment significantly reduces Tom20 oxidation, Bax mitochondrial recruitment, and GSDME cleavage, further confirming that ROS serves as the central regulator of this signaling pathway (47)

Although research on intranasal administration of BPC-157 is extremely limited, broad studies on the potential benefits of intranasally administered peptide therapeutics suggest that this may be a beneficial RoA [11, 12]

It is a foundational component of cellular resilience

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