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Prevention of oxidative damage and maintenance of mitochondrial function The neuroprotective behavior displayed by Sp intrigued a parallel study in the Drosophila eye since it displays detectable phenotypes in response to neurophysiological disruptions such as progressive loss of ommatidia accompanied with disrupted rhabdomere arrangement 38

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Literature on BPC-157 research includes model-specific work involving tendon fibroblast behavior, angiogenesis, nitric oxide signaling, endothelial context, and FAK-paxillin pathway discussion [4] [6] [9]

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