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Clinical, Cosmetic and Investigational Dermatology vol
Since PRDX2 has been shown to be glutathionylated by GSH at its cysteine residues to protect against its hyperperoxidation 41 , it is possible that elevated GSH levels in T315I-Bcr-Abl cells might prevent PRDX2 monomer formation, therefore abrogating ROS-induced replication stress
Data like this could play an important role in shifting how obesity treatment is viewedboth clinically and by insurers, Ali noted to MNT
However, there are two patterns of action of this compound: (I) ascorbic acid significantly increases the level of GSH without affecting or even reducing the activity of enzymes associated with this molecule (glutathione peroxidase, GSH-Px
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