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Convergent pathophysiological axes linking scar formation and AD Although scar formation and AD differ in clinical phenotype and onset context, they share a pathophysiological axis in which fibrosis is autonomously maintained and amplified at the level of the lesional microenvironment (Henderson et al

Because the total protein content of tissues was not affected by the d-carnitine treatment, it is also possible that ingested protein is digested more efficiently in these fish, such that amino acid absorption from the gut can be increased

Sima AA: Acetyl-L-carnitine in diabetic polyneuropathy: experimental and clinical data

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