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For instance, abundant evidence supports that cell shrinkage is mediated by K + efflux

Because 5-MTHF is a reduced form of folic acid that does not require conversion by dihydrofolate reductase, some direct effects may be attributable to redox mechanisms that are not seen when oral folic acid is used to increase plasma folate levels [58,59]

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Restoring its function via Caveolin-1 overexpression has been shown to inhibit ferroptosis by improving mitochondrial homeostasis, thereby mitigating cognitive decline both in vitro and in vivo (67)