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Beyond the nervous system, liraglutide intervention alleviates doxorubicin-induced testicular damage and cognitive dysfunction by counteracting oxidative injury and restoring PI3K/Akt/mTOR signaling homeostasis

These alterations may reflect underlying pathophysiological mechanisms of the disease [393, 441, 442]

The aim of the present narrative review was to describe the gastrointestinal adverse effects of the antidiabetic drugs, their pathophysiologic mechanisms, and the corresponding therapeutic measures

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