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The pretreatment with Ab reduced the systemic levels of inflammatory cytokines (TNF-, IL-1), increased the antioxidant response in several organs and tissues (GSH and TEAC) with concomitant inhibition of oxidative damage (lipid peroxidation in serum, peritoneal cavity, heart and liver) of septic mice

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FIGURE 3 3.3 GHK-Cu promoted mucosal healing GHK-Cu treatment significantly enhanced of ZO-1 and Occludin expression in colitic mice, which were downregulated by DSS (Figures 4AC)

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