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Description

Pro-inflammatory cytokines such as IL-1 and TNF produce toxicity through multiple pathways including complement activation, production of arachidonic acid metabolites and ROS formation (Neher et al., 2011)

The loss of GLP-1 effect on stimulating insulin secretion at basal plasma glucose concentrations limits the hypoglycemic risk even at high pharmacological concentrations of GLP-1 (28, 58)