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Studies examine: VEGFR2 activation pathways and intracellular adaptor protein recruitment Focal adhesion kinase (FAK) phosphorylation mechanisms Paxillin recruitment and integrin-actin cytoskeleton interactions Endothelial cell proliferation and migration assays in vitro Research models study the combined effects of VEGFR2-mediated proliferation and FAK-paxillin-mediated cellular motility in angiogenesis pathway investigations

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GHK-Cu increased production of nerve growth factor (NGF), neurotrophin-3 (NT-3), and neurotrophin-4 (NT-4), sped up regeneration of nerve fibers from nerve stubs, and increased axon count and proliferation of Schwann cells compared to controls