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These data strongly suggest the idea that resistance is not only due to a mutation in a target gene but also to a more wide cellular re-organization ranging from proteomic modulations as those observed in cell wall of resistant cells [24], [25] to metabonomic variations as those related to increased levels of intracellular GSH
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940 TQCN treatment may improve numerous neurodegenerative diseases related with brain iron accumulation and ameliorate severe cognitive impairment in AD mice due to its multiplex modulation of the pathogenic process that triggers ferroptosis
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