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The ability of metformin to increase GLP-1 levels independently of effects on DPP-4 suggests there could be a direct benefit in terms of incretin action by combining metformin with DPP-4 inhibitors

The 49-fold potency enhancement represents intrinsic receptor activation capacity rather than a prediction of clinical effect size

Are patients, whether they have a cancer diagnosis or not, are they coming to you and having trouble getting access to a GLP-1 receptor agonist and resorting to compounded agents

We discuss how ME/CFS emerges from an intricate web of genetic vulnerabilities and environmental triggers, notably viral infections, leading to a complex series of pathological responses including immune dysregulation, chronic inflammation, gut dysbiosis, and metabolic disturbances