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From a reproductive endocrine perspective, canonical PCOS is characterized by: (i) hyperandrogenism, presenting as hirsutism, acne, and androgenetic alopecia, with elevated total/free testosterone, an increased free androgen index (FAI), and occasionally elevated dehydroepiandrosterone sulfate (DHEAS)

Initiating a GLP-1 agent in a patient with an undiagnosed TSH abnormality or pre-existing hypoglycemia without that context carries meaningful clinical risk

Notably, QMP engineering ensures favorable pH-dependent FcRn binding, also in the absence of the C-terminal leucine residue

But these drugs dont work for everyone