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modelled a ROS-dependent mitochondrial oscillator, which included modelling the kinetics of glutathione peroxidase and reductase (Cortassa et al., 2004)

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1) was characterized in model proteins like human serum albumin and alkyl hydroperoxide reductase E from Mycobacterium tuberculosis ( Mt AhpE, a one-cysteine peroxiredoxin, particularly fast-reacting with hydroperoxides) ( explains the marked increases in persulfide levels observed in cell cultures treated with H 2 O 2 and H 2 S/HS ( The attack of HS on a disulfide (Eq

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