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During one experiment using a murine model, Pal-GHK was suggested to suppress the activation of Nuclear Factor kappa-light-chain-enhancer of activated B cells (NF-B) and p38 mitogen-activated protein kinase (MAPK) signaling pathways, both of which are associated with inflammation

In scientific research environments, it is used to examine molecular mechanisms of cell communication, angiogenesis, and signaling related to tissue organization

It also reduced the number of TGIF-positive neuronal cells, decreased the infarction volume, increased local blood flow, and ultimately improved functional outcomes in rats with TBI