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A therapeutic agent selected for the treatment of wounds should ideally improve one or more phases of healing without producing deleterious side effects
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To confirm that the observed effects were mediated through direct GLP-1R activation, or indeed, that they are not secondary autocrine effects of insulin, we performed 2-NBDG uptake experiments in the presence of the GLP1R antagonist Exendin (939) and the potassium channel opener, diazoxide
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It is a powerful antioxidant that has been shown to improve cognitive function, memory, and mood
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Neuroinflammation 18 , 106 (2021)
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