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Mitochondria as the specific site of intracellular melatonin production is generally supported by the published data and is consistent with the evolution of these organelles, which were derived from prokaryotes that presumably had the capacity of melatonin synthesis ( Figure 2 )

management of pre-existing comorbidities that can avoid clinical deterioration, readmission of patients and avoid other pathologies and symptoms development

they do not predict how a given medication will perform

Comparing the two requires stitching together separate trials that differ in sponsor, population, duration, and comparator a chain with several weak links