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Blocking GSH synthesis in combination with JMF4073 depletes dTTP/dCTP pools to eradicate T315I-Bcr-Abl leukemia We hypothesize that reducing the GSH level to increase replication stress can render T315I-Bcr-Abl leukemia more susceptible to JMF4073 in vivo

After pretreatment of thrombocytes from healthy subjects with collagen to mimic the preactivation in vitro , digoxin induced calcium mobilization, arachidonic acid release, TxB2 biosynthesis, platelet PAC-1 and P-selectin expression, and promoted platelet aggregation in a dose-dependent manner

Common research solvents include sterile water, bacteriostatic water (0.9% benzyl alcohol), and buffered saline at neutral pH

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