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Intra-articular injection of Cit-AuNRs@Anti-TRPV1 alleviates cartilage degradation by activating TRPV1 to inhibit ferroptosis in chondrocytes, reduces pain and improves physical activity in mice with medial meniscus instability-induced OA

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In keeping with the role of GPX3 as a stress-responsive antioxidant enzyme, there are binding sites at its promoter for hypoxia-inducible factor 1 (HIF-1), specificity protein 1 (Sp1) transcription factor, a metal response element (MRE) and an antioxidant response element (ARE) [20]