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An upregulation of KYAT1 and AADAT may reflect a compensatory astrocytic response to diminished KYNA availability, whereby upregulation of KAT enzymes and glutamate uptake mechanisms attempts to restore NMDAR inhibition and stabilize glutamatergic tone

The route can shift the GI picture (injectable avoids gut-driven complaints) without changing the underlying metabolism

Hoboken, NY: John Wiley & Sons, Ltd