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doi: 10.18502/ijrm.v17i9.5102 13

IGF-DES and Skeletal Muscle Hypertrophy Research IGF-1 signalling via the PI3K/Akt pathway drives protein synthesis through activation of ribosomal protein S6 and the translation initiation factor eIF4E downstream of mTORC1, and can suppress protein degradation via inhibition of FoxO-mediated transcription of E3 ubiquitin ligases mechanisms that are directly relevant to muscle hypertrophy and atrophy research models in which IGF-DES is employed as a research tool

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Gender differences in transformation Men and women experience somewhat different GLP-1 transformation patterns