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Trimethylamine N-oxide promotes apoE(-/-) mice atherosclerosis by inducing vascular endothelial cell pyroptosis via the SDHB/ROS pathway

The topological properties of the drug-disease network were analyzed using Cytoscape software, as demonstrated in Figure 5D

Nature Reviews Endocrinology , 5(7), 374381

The effects were modest and the clinical relevance uncertain, but they highlight that GHS-R1a signaling extends beyond the somatotroph axis and has the potential to influence multiple endocrine systems