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The activation of the classical cAMP pathway is achieved by releasing irritating G protein subunit Gs through the exchange of guanine nucleotide and the activation/dissociation of heterogeneous trimer G protein, and in pancreatic cells, GLP-1 binds to the specific guanine nucleotide-binding protein-coupled receptor GLP-1R, leading to adenylyl cyclase activation and elevated cAMP levels (Mayo et al., 2003

We assume that the flexibility of the polypeptide backbone and the targeting ligands could help the uptake and permeability of the 3-PLGs with similarly greater size at the BBB model

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Upper arm injections should be administered by a caregiver