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The latter option, that loss of oxidants can augment apoptosis, is supported by findings that correlate increased cellular hydrogen peroxide levels with resistance to apoptotic signaling in some cell systems (18, 86), as catalase overexpression in either cytoplasmic or mitochondrial compartments potentiated apoptosis, whereas inhibiting endogenous catalase promoted cell survival

69 In addition to the sirtuin family of deacetylases, other deacetylases and acetyltransferases are also strongly associated with aging, including HDAC1, 70 CBP-1, 71 KAT6A/B, 72 and SAS2

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