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While such regulatory steps are necessary, they remain insufficiently tailored to the specific risks faced by individuals with, or at risk of, eating disorders

GLP-1 (Glucagon-like peptide-1)...Acts on the hypothalamus (satiety center) in the brain to suppress appetite GIP (Glucose-dependent insulinotropic polypeptide)...Promotes the secretion of glucagon, which in addition to insulin, promotes fat breakdown and suppresses appetite Feeling full after a meal is due to the secretion of GLP-1 and GIP

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