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Results l-Carnitine attenuated the increased ROS levels in the cytosol and mitochondria of PFOS-treated RTCs and the cell death of such cells We previously demonstrated that N-acetylcysteine (NAC), a ROS scavenger, rescues RTCs from PFOS-mediated apoptosis triggered by autophagy activation because the autophagy inhibition due to 3-methyladenine can block the aforementioned apoptosis 3

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In addition, most joint structures, ligaments, and tendons are made of different kinds of collagen

Selenium is also an essential component of other metabolic systems that deal with oxidative stress, such as the thioredoxin system