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Combined with the structural changes to the Purkinje cells [179, 180], possible Purkinje cell loss [181], Purkinje cell axonal alterations, and/or abnormal Purkinje cell synaptic organization [182, 183] reported in postmortem human ET tissue, the VIM neurons are likely entrained by abnormal cerebellar activity

10.3390/ijms20010231 Int J Mol Sci

Published Scientific Research Peer-reviewed laboratory research investigating research peptides from leading scientific databases Structural and mechanistic insights into dual activation of cagrilintide in amylin and calcitonin receptors

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