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Abstract Chronic inflammation in type 2 diabetes (T2D), characterized by constitutively activated immune cells and elevated pro-inflammatory mediators along with hyperglycaemia and increased free fatty acids and branched chain amino acid levels, significantly alters the immuno-metabolic axis

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These rests of cells are sometimes referred to as benign metastases because these cell clusters may represent the end result of an intralymphatic migration of benign melanocytes