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Clinical features Beriberi: inadequate thiamine uptake due to malnutrition, heavy drinking, or increased demand (e.g., hyperthyroidism, pregnancy) Dry beriberi Symmetrical peripheral neuropathy (sensory and motor) Progressive muscle wasting Paralysis Confusion Wet beriberi High-output cardiac failure (due to systemic vasodilation) Dilated cardiomyopathy Cardiomegaly Edema Infantile beriberi: cardiomegaly, tachycardia, cyanosis, aseptic meningitis (vomiting and seizures) Wernicke encephalopathy Leigh syndrome Diagnostics: vitamin B 1 administration RBC transketolase activity In malnourished or alcohol-dependent patients, always administer thiamine before giving dextrose to decrease the risk of precipitating or exacerbating Wernicke encephalopathy

N Engl J Med 2017;377:1319-30

The most commonly used nonbiologic DMARDs are: Methotrexate Sulfasalazine Hydroxychloroquine Leflunomide DMARDs used in patients whose disease does not respond to the initial medications include: Azathioprine Cyclosporine

This mutation impairs splicing and cellular proliferation, albeit to a lesser extent than other SNIP1 FHA domain mutations that we identified as mediators of direct contacts with phosphorylated SF3B1