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Furthermore, it ameliorates hypertrophic cardiomyopathy and reverses diabetes-induced mitochondrial damage by restoring the expression and function of crucial cardiac energy metabolic proteins [37, 228]

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doi: 10.3390/genes16101166 361

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Astrocytes in AD model animals demonstrated up-regulated expression of genes (CPS1, OCT, ASL, ARG1, and ODC1) and metabolites (aspartate, ammonia, urea, putrescine, and GABA) of the urea cycle

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