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GLP-1R agonists have demonstrated the ability to improve cognitive function, reduce hippocampal synaptic loss, and decrease A plaque deposition and A oligomer levels in APP/PS1 transgenic mice [53, 54]
The demonstrated importance of protecting the terminals to pharmacokinetic characteristics suggests numerous additional synthetic approaches that may be applicable including the insertion of non-peptide linkages between the first and second amino acids, the replacement of the N~terminal amino acid with a non-a amino acid, and N-terminal acylation
Autoradiography revealed that autistic patients exhibited a significant decrease in the number of GABAA receptors and benzodiazepine binding sites in the fusiform gyrus (FFG) and posterior cingulate cortex (PCC), accompanied by an increased binding affinity at the surface of GABAA receptors
